Serveur d'exploration sur la maladie de Parkinson

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substantia nigra And NotFrance Javoy-Agid

List of bibliographic references

Number of relevant bibliographic references: 7.
Ident.Authors (with country if any)Title
000066 David A. Price [États-Unis] ; Alex A. Martinez [États-Unis] ; Alexandre Seillier [États-Unis] ; Wouter Koek [États-Unis] ; Yolanda Acosta [États-Unis] ; Elizabeth Fernandez [États-Unis] ; Randy Strong [États-Unis] ; Beat Lutz [Allemagne] ; Giovanni Marsicano [France] ; James L. Roberts [États-Unis] ; Andrea Giuffrida [États-Unis]WIN55,212‐2, a cannabinoid receptor agonist, protects against nigrostriatal cell loss in the 1‐methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine mouse model of Parkinson’s disease
000096 Andrew Lees (neurologue) [Royaume-Uni] ; Marianna Selikhova [Royaume-Uni] ; Luiz Augusto Andrade [Brésil] ; Charles Duyckaerts [France]The black stuff and Konstantin Nikolaevich Tretiakoff
000116 Vincent Paillé [France] ; Vincent Henry [France] ; Laurent Lescaudron [France] ; Philippe Brachet [France] ; Philippe Damier [France]Rat model of Parkinson's disease with bilateral motor abnormalities, reversible with levodopa, and dyskinesias
000184 Yassine Darbaky [France] ; Claude Forni [France] ; Marianne Amalric [France] ; Christelle Baunez [France]High frequency stimulation of the subthalamic nucleus has beneficial antiparkinsonian effects on motor functions in rats, but less efficiency in a choice reaction time task
000225 P. Damier ; E. C. Hirsch ; Yves Agid [France] ; A. M. GraybielThe substantia nigra of the human brain
000250 Brigitte Piallat [France] ; Abdelhamid Benazzouz [France] ; Alim-Louis Benabid [France]Subthalamic Nucleus Lesion in Rats Prevents Dopaminergic Nigral Neuron Degeneration After Striatal 6‐OHDA Injection: Behavioural and Immunohistochemical Studies
000254 A. Mouatt-Prigent [France] ; J. O. Karlsson [Suède] ; Yves Agid [France] ; E. C. Hirsch [France]Increased m-calpain expression in the mesencephalon of patients with parkinson's disease but not in other neurodegenerative disorders involving the mesencephalon: a role in nerve cell death?

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